Emily Cybulla

Emily Cybulla's research focuses on how cells respond to DNA damage during replication, particularly in cancers with defects in DNA repair genes like BRCA1. Her work examines molecular pathways (such as PRIMPOL, RAD18, and GAS6/AXL signaling) that allow cancer cells to tolerate replication stress, and explores how targeting these pathways with drugs like PARP inhibitors, chemotherapy, or other agents could make cancer treatment more effective. Much of her research uses laboratory models of breast, ovarian, and prostate cancers to study these mechanisms and their potential clinical applications.

Publication output has grown from roughly zero to one paper per year in the late 2010s to a steadier pace of about two to five papers annually in recent years, suggesting increasing research activity over the past decade.

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