Publications
277
Citations
10,065
Est. group size
~23
Recurring co-author estimate
Active years
40
Publishing since 1987
Sándor Györke's research focuses on how heart muscle cells manage calcium signaling and how disruptions in this process lead to irregular heartbeats (arrhythmias) and heart failure. His work examines specific proteins like calmodulin and ryanodine receptors that control calcium flow within heart cells, as well as how mitochondria (the cell's energy centers) interact with this calcium signaling system. This research aims to understand the molecular mechanisms behind inherited and acquired heart rhythm disorders, potentially informing future therapies.
Publication output peaked around 2020-2021, then declined through 2023-2024, with a modest rebound in 2025-2026, though the last-five-year average is lower than earlier in the decade.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- BPS2026 – Dynamic-structure redesign of calmodulin reduces Ca2+ leak and reveals mechanistic insights into RyR2 regulation
Biophysical Journal · 2026
- BPS2026 – Sub-sarcomeric spatial multiomics reveals distinct organization of excitation-contraction coupling mRNA translation
Biophysical Journal · 2026
- BPS2026 – The D73N troponin C mouse: A translational model of dilated cardiomyopathy with mitochondrial ROS and novel atrial myxomas
Biophysical Journal · 2026
- Dynamic-Structure Redesign of Calmodulin Reveals Mechanistic Constraints on Ryr2 Regulation
bioRxiv (Cold Spring Harbor Laboratory) · 2026
- Restoration of mitochondrial Ca2+ and redox homeostasis by enhancement of SK channels rescues Ca2+ cycling in HFpEF cardiomyocytes
Journal of Molecular and Cellular Cardiology · 2026
- Disruption of Localized Protein Synthesis in the Sarcoplasmic Reticulum Impairs Cardiac Excitation-Contraction Coupling in Heart Failure
Journal of Molecular and Cellular Cardiology Plus · 2026
- Pharmacological Enhancement of Small Conductance Ca <sup>2+</sup> -Activated K <sup>+</sup> Channels Suppresses Cardiac Arrhythmias in a Mouse Model of Catecholaminergic Polymorphic Ventricular Tachycardia
Circulation Research · 2025
- BPS2025 - Spatial compartmentalization of the expression of CaM genes supports versatile Ca2+-CaM signaling in cardiac myocytes
Biophysical Journal · 2025
- BPS2025 - Fractional recovery of calmodulin, troponin C, and their target protein's mRNA in cardiomyocytes and fibroblasts
Biophysical Journal · 2025
- BPS2025 - Localized synthesis of cardiac excitation-contraction coupling proteins: A dual role for the sarcoplasmic reticulum
Biophysical Journal · 2025
- Increased Intermembrane Space [Ca <sup>2+</sup> ] Drives Mitochondrial Structural Damage in CPVT
Circulation Research · 2025
- BPS2025 - Mechanisms underlying distinct cardiac arrhythmia phenotypes caused by calmodulin mutation D132E in Calm2 and Calm3 genes
Biophysical Journal · 2025
- Increased Intermembrane Space [Ca2+] Drives Mitochondrial Structural Damage in CPVT
The Medicine Forum · 2025
- Distinct intracellular spatiotemporal expression of Calmodulin genes underlies functional diversity of Calmodulin-dependent signalling in cardiac myocytes
Cardiovascular Research · 2025
- Reduced mitochondria electron transport chain supercomplex assembly contributes to calcium-dependent ventricular arrhythmia in cardiac hypertrophy
Biophysical Journal · 2024
- Biophysical Journal×42
- Circulation×10
- Basic Research in Cardiology×4
- The Journal of General Physiology×4
- Microscopy and Microanalysis×4
- Isabelle Deschênes
Medicine · The Ohio State University
- Xiaoping Wan
Medicine · The Ohio State University
- Vladimir Bogdanov
Medicine · The Ohio State University
- Radmila Terentyeva
Medicine · The Ohio State University
- Dmitry Terentyev
Medicine · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile