Rajgopal Govindarajan
Biochemistry, Genetics and Molecular Biology · The Ohio State University
Publications
155
Citations
2,911
Est. group size
~14
Recurring co-author estimate
Active years
25
Publishing since 2002
This researcher studies molecular mechanisms underlying pancreatic cancer, with a particular focus on how signaling proteins that regulate G-protein pathways (a class of molecules involved in relaying signals inside cells) contribute to tumor growth and progression. Recent work centers on how the loss of a specific regulatory protein, RGS11, promotes cancer-promoting features in pancreatic tumors. The lab's outputs also touch on related topics such as epigenetics (chemical modifications controlling gene activity), RNA changes in cancer, and kinase signaling (enzyme-driven cell communication pathways).
Publication output has grown substantially in recent years, with sharp increases in 2022-2023 and again in 2026 compared to a slower, low-output period from 2017 to 2021.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Supplementary Figure S4 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S5 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S6 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Figure S1 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Figure S2 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Figure S8 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Figure S7 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S2 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Materials & Methods from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Figure S5 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Data from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S7 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S1 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S3 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Supplementary Table S12 from Loss of Regulator of G Protein Signaling 11 Promotes Protumorigenic Features in Pancreatic Cancer
2026
- Nature Communications×5
- Journal of Pharmacology and Experimental Therapeutics×4
- Cancer Research×4
- Molecular Cancer Research×3
- Journal of the American Society of Nephrology×3
- Joo Mi Yi
Biochemistry, Genetics and Molecular Biology · Indiana University
- Samantha L. Tinsley
Biochemistry, Genetics and Molecular Biology · Purdue University West Lafayette
- Rulong Shen
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Ilaria Cosentini
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Heather M. O’Hagan
Biochemistry, Genetics and Molecular Biology · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
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