Heather M. O’Hagan
Biochemistry, Genetics and Molecular Biology · Indiana University
Publications
220
Citations
3,316
Est. group size
—
Recurring co-author estimate
Active years
26
Publishing since 2001
Heather M. O'Hagan studies the molecular and epigenetic changes that drive cancer, focusing on how genes get switched off through DNA methylation and chromatin-modifying proteins (such as Polycomb/CBX7). Her work spans colorectal and breast cancers, examining processes like heritable gene silencing, metastasis, drug resistance, and the role of immune cells and extracellular vesicles in tumor biology.
Publication output was steady at roughly 5-9 per year through 2022, then rose sharply, with recent years averaging over 30 per year (a 2023 spike partly reflects many supplementary-material records).
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- Mast Cells Selectively Deliver Extracellular Vesicle‐Encapsulated <scp>mRNA</scp> to Colorectal Cancer Cells
Immunology and Cell Biology · 2026
- Tumor-intrinsic role of ICAM-1 in driving metastatic progression of triple-negative breast cancer through direct interaction with EGFR
Molecular Cancer · 2024
- Supplementary Table 1 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 5 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Table 2 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 4 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 3 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 2 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 1 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Tables 1-3 from Frequent Inactivation of <i>Cysteine Dioxygenase Type 1</i> Contributes to Survival of Breast Cancer Cells and Resistance to Anthracyclines
2023
- Supplementary Figures 1-3 from Frequent Inactivation of <i>Cysteine Dioxygenase Type 1</i> Contributes to Survival of Breast Cancer Cells and Resistance to Anthracyclines
2023
- Supplementary Table 1 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 1 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Table 2 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Supplementary Figure 4 from Polycomb CBX7 Promotes Initiation of Heritable Repression of Genes Frequently Silenced with Cancer-Specific DNA Hypermethylation
2023
- Cancer Research×17
- bioRxiv (Cold Spring Harbor Laboratory)×12
- Cancers×4
- PMC×4
- Molecular Cancer Research×3
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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