Philip N. Tsichlis
Biochemistry, Genetics and Molecular Biology · The Ohio State University
Publications
317
Citations
26,274
Est. group size
~10
Recurring co-author estimate
Active years
48
Publishing since 1977
Philip N. Tsichlis studies the molecular biology of cancer, focusing on how signaling proteins like AKT (a kinase involved in cell growth and survival) and epigenetic regulators (proteins that control gene activity without changing DNA sequence) contribute to tumor development in cancers such as melanoma, breast cancer, and blood cell malignancies. Much of the work uses mouse models to test how removing or altering specific genes affects tumor formation and spread. Prospective students would likely engage with cancer cell biology, mouse genetics, and signaling pathway research.
Publication output has fluctuated over the last decade with a notable spike in 2023, following a period of moderate and variable annual output.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- AKT2 Loss Impairs BRAF-Mutant Melanoma Metastasis
Cancers · 2023
- AKT2 Loss Impairs BRAF-Mutant Melanoma Metastasis
bioRxiv (Cold Spring Harbor Laboratory) · 2023
- Data from A Requirement for Cyclin-Dependent Kinase 6 in Thymocyte Development and Tumorigenesis
2023
- Supplementary Figures 1-5 from A Requirement for Cyclin-Dependent Kinase 6 in Thymocyte Development and Tumorigenesis
2023
- Data from A Requirement for Cyclin-Dependent Kinase 6 in Thymocyte Development and Tumorigenesis
2023
- Supplementary Figures 1-5 from A Requirement for Cyclin-Dependent Kinase 6 in Thymocyte Development and Tumorigenesis
2023
- Supplementary Material, Methods and Figure Legend from Akt1 Ablation Inhibits, whereas Akt2 Ablation Accelerates, the Development of Mammary Adenocarcinomas in Mouse Mammary Tumor Virus (MMTV)-ErbB2/Neu and MMTV-Polyoma Middle T Transgenic Mice
2023
- Supplementary Material, Methods and Figure Legend from Akt1 Ablation Inhibits, whereas Akt2 Ablation Accelerates, the Development of Mammary Adenocarcinomas in Mouse Mammary Tumor Virus (MMTV)-ErbB2/Neu and MMTV-Polyoma Middle T Transgenic Mice
2023
- Supplementary Figure 1 from Akt1 Ablation Inhibits, whereas Akt2 Ablation Accelerates, the Development of Mammary Adenocarcinomas in Mouse Mammary Tumor Virus (MMTV)-ErbB2/Neu and MMTV-Polyoma Middle T Transgenic Mice
2023
- Data from Akt1 Ablation Inhibits, whereas Akt2 Ablation Accelerates, the Development of Mammary Adenocarcinomas in Mouse Mammary Tumor Virus (MMTV)-ErbB2/Neu and MMTV-Polyoma Middle T Transgenic Mice
2023
- Supplementary Figure 1 from Akt1 Ablation Inhibits, whereas Akt2 Ablation Accelerates, the Development of Mammary Adenocarcinomas in Mouse Mammary Tumor Virus (MMTV)-ErbB2/Neu and MMTV-Polyoma Middle T Transgenic Mice
2023
- Data from Akt1 Ablation Inhibits, whereas Akt2 Ablation Accelerates, the Development of Mammary Adenocarcinomas in Mouse Mammary Tumor Virus (MMTV)-ErbB2/Neu and MMTV-Polyoma Middle T Transgenic Mice
2023
- Supplementary Figure 2 from NDY1/KDM2B Functions as a Master Regulator of Polycomb Complexes and Controls Self-Renewal of Breast Cancer Stem Cells
2023
- Supplementary Figure 7 from NDY1/KDM2B Functions as a Master Regulator of Polycomb Complexes and Controls Self-Renewal of Breast Cancer Stem Cells
2023
- Supplementary Materials and Methods, Figure Legends, Tables 1 - 5 from NDY1/KDM2B Functions as a Master Regulator of Polycomb Complexes and Controls Self-Renewal of Breast Cancer Stem Cells
2023
- bioRxiv (Cold Spring Harbor Laboratory)×11
- Cancer Research×11
- Cancers×3
- Proceedings of the National Academy of Sciences×3
- The Journal of Immunology×3
- Min Chen
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Ilaria Cosentini
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Kenneth P. Nephew
Biochemistry, Genetics and Molecular Biology · Indiana University
- Heather M. O’Hagan
Biochemistry, Genetics and Molecular Biology · Indiana University
- Ming Li
Biochemistry, Genetics and Molecular Biology · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile