Publications
84
Citations
969
Est. group size
—
Recurring co-author estimate
Active years
22
Publishing since 2005
Typically publishes in teams of ~7 · 13% small-team papers (≤3 authors) · across 34 venues
- Molecular dynamics of Brodmann Area 22 in development and autism
bioRxiv (Cold Spring Harbor Laboratory) · 2026
- The α-MSH-MC1R Axis Modulates Sex-Specific Senescence and Inflammation Processes in Human Articular Chondrocytes and Mice Knee Joints
University of Regensburg Publication Server (University of Regensburg) · 2026
- Dissecting the Impact of α-MSH-MC1R-cAMP Signaling on UVA-Induced Stress in Fibroblasts - Implications for Regulation of Cutaneous Photoaging
Aging and Disease · 2026
- Mindestanforderungen für die sichere Applikation von medikamentöser Tumortherapie durch onkologische Pflegefachpersonen
Onkologische Pflege. · 2026
- Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Cell Communication and Signaling · 2026
- Additional file 3 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 2 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 2 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 4 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 1 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 3 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 1 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Additional file 4 of Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Multi-omics and experimental evidence in human chondrocytes identify caspase-8 as a non-apoptotic regulator of inflammatory, senescent, and fibrotic signaling in osteoarthritis
Figshare · 2026
- Figshare×10
- Molecular Immunology×6
- Frontiers in Immunology×4
- Immunobiology×4
- bioRxiv (Cold Spring Harbor Laboratory)×4
- Sudha Agarwal
Medicine · The Ohio State University
- Jonathan Bowyer
Medicine · Purdue University West Lafayette
- Biming Wu
Medicine · University of Michigan
- Sonali Karnik
Medicine · Indiana University
- Alexander J. Knights
Medicine · University of Michigan
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 25, 2026.
Claim or correct this profile