Liangguang Leo Lin

Biochemistry, Genetics and Molecular Biology · University of Michigan

Early career · publishing since 2019Rising activity

Endoplasmic Reticulum Stress and Disease Mitochondrial Function and Pathology Autophagy in Disease and Therapy Pancreatic function and diabetes Cellular transport and secretion

Publications

Citations

418

Est. group size

Recurring co-author estimate

Active years

Publishing since 2019

In context

Among 543 early career Biochemistry, Genetics and Molecular Biology PIs within our index

Citations: 418cohort median 176

Publications / year (recent): 5.2cohort median 2.4

Typically publishes in teams of ~9 · 0% small-team papers (≤3 authors) · across 6 venues

Publication cadence

Publications per year over the last 10 years — averaging 5.2/year recently

Collaborators per year

Distinct co-authors at University of Michigan each year — a rough indication of whether the group around this researcher has been growing or contracting. An estimate from co-authorship, not a roster: collaborators are not necessarily lab members, and recent years can be under-counted while indexing catches up.

Recent publications

Structural basis and pathological implications of the dimeric OS9-SEL1L-HRD1 ERAD Core Complex
Nature Communications · 2026
Functional integrity of the SEL1L–HRD1 complex is critical for endoplasmic reticulum–associated degradation and organismal viability
Proceedings of the National Academy of Sciences · 2026
SEL1L-HRD1 ER-associated degradation facilitates prohormone convertase 2 maturation and glucagon production in islet α cells
Nature Communications · 2026
A molecular switch in NAC prevents mitochondrial protein mistargeting by SRP
Nature Communications · 2026
The endoplasmic reticulum-associated degradation machinery selectively degrades stress-induced TIN1 during stress recovery
PLANT PHYSIOLOGY · 2025
Requirements for nuclear GRP78 transcriptional regulatory activities and interaction with nuclear GRP94
Journal of Biological Chemistry · 2025
Structural basis and pathological implications of the dimeric OS9-SEL1L-HRD1 ERAD Core Complex
bioRxiv (Cold Spring Harbor Laboratory) · 2025
A molecular switch in NAC prevents mitochondrial protein mistargeting by SRP
bioRxiv (Cold Spring Harbor Laboratory) · 2025
Functional rescue of a fatal ERAD mutation via alternative splicing
bioRxiv (Cold Spring Harbor Laboratory) · 2025
Functional integrity of the SEL1L-HRD1 complex is critical for ERAD and organismal viability
bioRxiv (Cold Spring Harbor Laboratory) · 2025
Genome-wide screens identify SEL1L as an intracellular rheostat controlling collagen turnover
Nature Communications · 2024
Joint Detection Method for Two-Way Automatic Communication System
2024
Distribution Terminal Clock Synchronization Method Using Industrial Frequency Voltage Signals
2024
Biallelic Cys141Tyr variant of SEL1L is associated with neurodevelopmental disorders, agammaglobulinemia, and premature death
Journal of Clinical Investigation · 2023
Selective degradation of an ER stress-induced protein by ER-associated degradation mechanism during stress recovery
bioRxiv (Cold Spring Harbor Laboratory) · 2022

Publishes in

Nature Communications×9
bioRxiv (Cold Spring Harbor Laboratory)×9
Journal of Clinical Investigation×3
Nature Cell Biology×1
JCI Insight×1

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This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.

Last updated Jul 25, 2026.

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