Established · publishing since 1989Rising activity
Pancreatic and Hepatic Oncology ResearchEpigenetics and DNA MethylationFibroblast Growth Factor ResearchCancer, Hypoxia, and MetabolismCancer Cells and Metastasis
Publications
88
Citations
2,148
Est. group size
~8
Recurring co-author estimate
Active years
38
Publishing since 1989
In context
Among 4,953 established Medicine PIs within our index
Citations: 2,148p25 789 · p75 7,423 · cohort median 2,544
Publications / year (recent): 10.4p25 2 · p75 10.6 · cohort median 4.8
Typically publishes in teams of ~19 · 5% small-team papers (≤3 authors) · across 18 venues
Publication cadence
Publications per year over the last 10 years — averaging 10.4/year recently
Collaborators per year
Distinct co-authors at University of Michigan each year — a rough indication of whether the group around this researcher has been growing or contracting. An estimate from co-authorship, not a roster: collaborators are not necessarily lab members, and recent years can be under-counted while indexing catches up.
Recent publications
- Hypoxia-Induced Fibroblast IL-6 Promotes Immunosuppressive Macrophage Phenotypes in Pancreatic Cancer
Cells · 2026
- A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
Cancer Research · 2024
- Age- and ethnic-driven molecular and clinical disparity of East Asian breast cancers
BMC Medicine · 2024
- Data from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Age and ethnic-driven molecular and clinical disparity of East Asian breast cancers
Research Square · 2024
- Analysis of Response and Progression Patterns of Tyrosine Kinase Inhibitors in Recurrent or Metastatic Adenoid Cystic Carcinoma: A Post Hoc Analysis of Two KCSG Phase II Trials
Cancer Research and Treatment · 2024
- Supplementary Figure S9 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S2 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S4 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S3 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S12 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S6 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S8 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S10 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
- Supplementary Figure S1 from A Histone Methylation–MAPK Signaling Axis Drives Durable Epithelial–Mesenchymal Transition in Hypoxic Pancreatic Cancer
2024
Publishes in
- Cancer Research×6
- Cancer Research and Treatment×2
- bioRxiv (Cold Spring Harbor Laboratory)×2
- Cancer Immunology Research×2
- Nature Cell Biology×1
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This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 25, 2026.
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