Weinian Shou
Biochemistry, Genetics and Molecular Biology · Indiana University
Publications
302
Citations
8,621
Est. group size
—
Recurring co-author estimate
Active years
35
Publishing since 1992
Weinian Shou studies the molecular and genetic basis of heart development and heart disease. This work spans how cardiac muscle cells form and mature, the gene mutations behind congenital heart defects and inherited cardiomyopathies (such as desmoplakin- and lamin-related conditions), and how inflammation and signaling pathways contribute to cardiac injury and scarring. Much of the research uses mouse models and patient-derived stem cells to link specific genes to heart function.
Publication output was high in the late 2010s (around 16 per year) and has settled to a lower, steadier level of roughly 5 per year in recent years.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- iNKT cell activation exacerbates isoproterenol-induced cardiac injury through macrophage IFN-γ–STAT1 signaling
Life Sciences · 2026
- Essential role of NONO-HOXA1-Wnt axis in cardiomyocyte differentiation
Nature Communications · 2026
- PO-02-317 ESTABLISHING CLINICALLY RELEVANT LAMIN A/C MUTATION-MEDIATED CARDIOMYOPATHY MODELS WITH PATIENT-SPECIFIC IPSCS BY INCREASING LAMIN A/C TO B RATIOS AS SEEN IN HUMAN CARDIAC AGING HEARTS
Heart Rhythm · 2026
- Tunneling nanotube–like structures regulate distant cellular interactions during heart formation
Science · 2025
- Interpenetrating network hydrogel-loaded embryonic stem cell-derived endocardial cells improves cardiac function after myocardial infarction
Journal of Translational Medicine · 2025
- Abstract Thu149: Pathogenic desmoplakin variants induce long cell-membrane caveolin-1 (CAV1) cluster formation and decrease CAV1-mediated TGFβ1 receptor endocytosis via beclin-1 deficiency
Circulation Research · 2025
- Adipocyte‐specific ablation of plakoglobin in mice does not affect adiposity but results in sexual‐dimorphic effects on weight gain
Physiological Reports · 2025
- Targeting S100A9 Prevents β-Adrenergic Activation–Induced Cardiac Injury
Inflammation · 2024
- Disorganized chromatin hierarchy and stem cell aging in a male patient of atypical laminopathy-based progeria mandibuloacral dysplasia type A
Nature Communications · 2024
- Progresses in genetic testing in congenital heart disease
Medicine Plus · 2024
- PO-05-197 A TRUNCATED DESMOPLAKIN WITH ONLY VIMENTIN-BINDING DOMAINS CAN AMELIORATE PATHOLOGICAL FIBROGENESIS IN CARDIAC FIBROBLASTS WITH DSP VARIANTS AND HAPLOINSUFFICIENCY
Heart Rhythm · 2024
- Prenatal Inflammatory Exposure Predisposes Offspring to Chronic Kidney Diseases Via the Activation of the eIF2α-ATF4 Pathway
Inflammation · 2024
- Desmoplakin mutations in cardiac fibroblasts cause TGFβ1-mediated pathological fibrogenesis in desmoplakin cardiomyopathy via beclin-1 regulation
bioRxiv (Cold Spring Harbor Laboratory) · 2024
- Newly Identified Genes Linked to Tetralogy of Fallot
Phenomics · 2024
- Dbh+ catecholaminergic cardiomyocytes contribute to the structure and function of the cardiac conduction system in murine heart
Nature Communications · 2023
- Figshare×14
- PMC×10
- Nature Communications×4
- Scientific Reports×4
- bioRxiv (Cold Spring Harbor Laboratory)×4
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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