Publications
301
Citations
1,503
Est. group size
—
Recurring co-author estimate
Active years
22
Publishing since 2004
This researcher studies neurofibromatosis type 1 (NF1), a genetic condition that causes tumors to grow on nerves, using mouse models to understand how these tumors form and progress. The work explores the biology behind tumor growth, including how the neurofibromin protein and immune cells contribute to disease, and tests potential treatments such as recombinant proteins to slow tumor growth.
Publication activity was modest through 2021, absent in 2022, then rose dramatically from 2023 onward, indicating a sharp increase in recent output.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- Recombinant Human Neuregulin1‐β1 Significantly Reduces Schwannoma Growth in Mice
Annals of Neurology · 2025
- Irradiation of <i>Nf1</i> mutant mouse models of spinal plexiform neurofibromas drives pathologic progression and decreases survival
Neuro-Oncology Advances · 2021
- TMOD-06. MALIGNANT TRANSFORMATION OF PLEXIFORM NEUROFIBROMAS IN AN NF1 MUTANT MOUSE MODEL OF SPINAL IRRADIATION
Neuro-Oncology · 2020
- <i>Nf1</i><sup>+/−</sup>monocytes/macrophages induce neointima formation via CCR2 activation
Human Molecular Genetics · 2016
- Neurofibromin is a novel regulator of Ras-induced reactive oxygen species production in mice and humans
Free Radical Biology and Medicine · 2016
- Neurofibromin is a novel regulator of Ras-induced reactive oxygen species production in mice and humans
PMC · 2016
- Nf1+/- monocytes/macrophages induce neointima formation via CCR2 activation
PMC · 2016
- PMC×5
- Human Molecular Genetics×4
- Clinical Cancer Research×4
- Journal of Biological Chemistry×2
- Neuro-Oncology×2
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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