Publications
44
Citations
842
Est. group size
—
Recurring co-author estimate
Active years
16
Publishing since 2011
Tejinder Pal Khaket's research focuses on cancer biology, particularly on how cancer cells respond to stress and die, including processes like ferroptosis (an iron-dependent form of cell death), autophagy (a cellular self-degradation process), and mitochondrial dysfunction. Recent work examines combination drug therapies for cancers such as melanoma, thyroid cancer, and glioma, often targeting mutated genes like BRAFV600E alongside cell-death pathways to enhance treatment effectiveness. The work spans laboratory experiments in cell lines and preclinical models to identify potential drug combination strategies.
Publication output has increased notably in the most recent two years (2025-2026) after a period of lower, steady output earlier in the decade.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Dual targeting of BRAFV600E and ferroptosis results in synergistic anticancer activity via iron overload and enhanced oxidative stress
Journal of Experimental & Clinical Cancer Research · 2026
- Mitochondrial reverse electron transport regulates glioma stemness through Sirt3-HIF1α-SOX2 signaling
Cancer Letters · 2026
- Additional file 1 of Dual targeting of BRAFV600E and ferroptosis results in synergistic anticancer activity via iron overload and enhanced oxidative stress
Open MIND · 2026
- Dual targeting of BRAFV600E and ferroptosis results in synergistic anticancer activity via iron overload and enhanced oxidative stress
Figshare · 2026
- Dual targeting of BRAFV600E and ferroptosis results in synergistic anticancer activity via iron overload and enhanced oxidative stress
Figshare · 2026
- Additional file 1 of Dual targeting of BRAFV600E and ferroptosis results in synergistic anticancer activity via iron overload and enhanced oxidative stress
Figshare · 2026
- Dual targeting of PDPK1 and BRAF V600E is synthetically lethal
bioRxiv (Cold Spring Harbor Laboratory) · 2026
- Abstract 5708: Dual targeting of PDPK1 and mutated BRAFV600E is synthetically lethal
Cancer Research · 2026
- Combination nitazoxanide and auranofin treatment has synergistic anticancer activity in anaplastic thyroid cancer through enhanced activation of oxidative stress that leads to apoptosis
Cancer Letters · 2025
- Dual Targeting of <i> BRAF <sup>V600E</sup> </i> and Ferroptosis Results in Synergistic Anticancer Activity via Iron Overload and Enhanced Oxidative Stress
bioRxiv (Cold Spring Harbor Laboratory) · 2025
- Corrigendum to “Combination nitazoxanide and auranofin treatment has synergistic anticancer activity in anaplastic thyroid cancer through enhanced activation of oxidative stress that leads to apoptosis” [Cancer Letters 633 (2025) 217990]
Cancer Letters · 2025
- Anaplastic thyroid cancer spheroids as preclinical models to test therapeutics
Journal of Experimental & Clinical Cancer Research · 2024
- Ribosome stalling during <i>c-myc</i> translation presents actionable cancer cell vulnerability
PNAS Nexus · 2024
- Reverse electron transfer is activated during aging and contributes to aging and age‐related disease
EMBO Reports · 2023
- Deregulation of ER-mitochondria contact formation and mitochondrial calcium homeostasis mediated by VDAC in fragile X syndrome
Developmental Cell · 2023
- Cancer Letters×3
- Figshare×3
- Journal of Experimental & Clinical Cancer Research×2
- bioRxiv (Cold Spring Harbor Laboratory)×2
- Cancer Research×2
- Hong Li
Medicine · The Ohio State University
- Abtar Mishra
Medicine · Purdue University West Lafayette
- Abdalla Elbialy
Medicine · The Ohio State University
- Bhawana Bissa
Medicine · The Ohio State University
- Hsuan-Yeh Pan
Medicine · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile