Publications
124
Citations
6,602
Est. group size
—
Recurring co-author estimate
Active years
32
Publishing since 1993
Takeshi Kurita studies the biology of the female reproductive tract, focusing on how the uterus, cervix, ovaries, and vagina develop and how they can go wrong, including uterine fibroids (leiomyomas), ovarian tumors, and structural birth defects like imperforate vagina. This work relies heavily on mouse models to understand the cell biology and hormone signaling (such as estrogen) underlying these conditions, with an eye toward improving diagnosis and treatment of reproductive disorders. The research combines basic developmental biology with disease-oriented studies relevant to gynecology and cancer.
Output was modest and variable from 2017-2022 (0-7 papers/year) with a large spike in 2023 (33 items, likely including many supplementary files from a few major papers), suggesting an uneven but currently active publication pattern.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- High incidence of imperforate vagina in ADGRA3-deficient mice
BMC Biology · 2024
- Cervix: Cell Biology
Elsevier eBooks · 2024
- Data from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Table S1 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S2 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S4 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S4 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Table S1 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S1 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Data from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S3 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S2 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S3 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Figure S1 from Subtype-Specific Tumor-Associated Fibroblasts Contribute to the Pathogenesis of Uterine Leiomyoma
2023
- Data from Constitutive Activation of PI3K in Oocyte Induces Ovarian Granulosa Cell Tumors
2023
- Differentiation×5
- bioRxiv (Cold Spring Harbor Laboratory)×3
- Cell Death and Differentiation×2
- Cancer Research×2
- Endocrine Related Cancer×2
- Li Chen
Medicine · The Ohio State University
- David Wininger
Medicine · The Ohio State University
- Christina R. Ferreira
Medicine · Purdue University West Lafayette
- Augustine T. Peter
Medicine · Purdue University West Lafayette
- Janice P. Evans
Medicine · Purdue University West Lafayette
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
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