Publications
573
Citations
2,771
Est. group size
~2
Recurring co-author estimate
Active years
28
Publishing since 1999
Steven P. Angus studies cancer biology with a focus on how tumors respond to targeted drug treatments, particularly cancers driven by faulty cell-signaling pathways (such as those involving RAS and MEK proteins). Much of the work explores combination therapies and drug-resistance mechanisms in pediatric and other cancers including rhabdomyosarcoma, leukemia, osteosarcoma, and sarcomas. The research aims to identify drug combinations that shrink tumors or make cancer cells more vulnerable to treatment.
Publication output was modest and steady from 2017 to 2022 (roughly 4-12 per year), then sharply increased from 2023 onward, likely reflecting a change in output or record-keeping.
Generated by claude-opus-4-8 from public bibliographic data · Jul 9, 2026
Current awards run through January 2028 — about 1 year of funding on record from today. Awards are often renewed, so this is what is currently public, not a forecast.
Identification of novel therapeutic combinations for NF2 schwannomas
Matched to public NIH RePORTER and NSF records by name and institution. Awards from other agencies are not shown, and a match is not always found — this list may be incomplete.
Typically publishes in teams of ~15 · 1% small-team papers (≤3 authors) · across 37 venues
- Proteomic approaches for interrogating kinase signaling networks
Journal of Investigative Dermatology · 2026
- Supplementary Table 9 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Supplementary Table S5 from Efficacy of the Allosteric MEK Inhibitor Trametinib in Relapsed and Refractory Juvenile Myelomonocytic Leukemia: a Report from the Children’s Oncology Group
2026
- Supplementary Data from PLK1 Inhibition Induces Synthetic Lethality in Fanconi Anemia Pathway–Deficient Acute Myeloid Leukemia
2026
- Supplementary Data from PLK1 Inhibition Induces Synthetic Lethality in Fanconi Anemia Pathway–Deficient Acute Myeloid Leukemia
2026
- Supplementary Table S2 from Efficacy of the Allosteric MEK Inhibitor Trametinib in Relapsed and Refractory Juvenile Myelomonocytic Leukemia: a Report from the Children’s Oncology Group
2026
- Supp fig 2 from PLK1 Inhibition Induces Synthetic Lethality in Fanconi Anemia Pathway–Deficient Acute Myeloid Leukemia
2026
- Supp fig 3 from PLK1 Inhibition Induces Synthetic Lethality in Fanconi Anemia Pathway–Deficient Acute Myeloid Leukemia
2026
- Supplementary Figure S6 from Efficacy of the Allosteric MEK Inhibitor Trametinib in Relapsed and Refractory Juvenile Myelomonocytic Leukemia: a Report from the Children’s Oncology Group
2026
- Supplementary Table 8 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Supplementary Table 8 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Supplementary Table 8 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Supplementary Table 9 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Supplementary Table 9 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Supplementary Table 8 from Combined Inhibition of HRAS and MEK Induces Tumor Regression and Restores Myogenic Differentiation in HRAS-Mutant Rhabdomyosarcoma
2026
- Cancer Research×17
- UNC Libraries×14
- Neuro-Oncology×13
- bioRxiv (Cold Spring Harbor Laboratory)×5
- Clinical Cancer Research×4
- D. Wade Clapp
Medicine · Indiana University
- Li Jiang
Medicine · Indiana University
- Waylan Bessler
Medicine · Indiana University
- Abbi E. Smith
Medicine · Indiana University
- Eric Hawley
Medicine · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Sep 1, 2026.
Claim or correct this profile