Publications
88
Citations
752
Est. group size
~1
Recurring co-author estimate
Active years
9
Publishing since 2018
Silpa Gampala studies cancer biology, with a focus on identifying and targeting specific proteins and enzymes that cancer cells depend on. Much of the work examines pancreatic cancer and nerve-sheath tumors, exploring molecular targets such as the DNA-repair enzyme APE1/Ref-1 to block tumor growth, spread (metastasis), and resistance to therapy. The research combines laboratory studies of tumor mechanisms with early-stage drug discovery.
Publication activity was modest through the late 2010s and early 2020s but rose sharply in 2025 and 2026, indicating a strong recent increase in output.
Generated by claude-opus-4-8 from public bibliographic data · Jul 9, 2026
Typically publishes in teams of ~15 · 3% small-team papers (≤3 authors) · across 25 venues
- Additional file 1 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 5 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 6 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 7 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 2 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 3 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 6 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 4 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 1 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 5 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 7 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 2 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Additional file 4 of Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Figshare · 2026
- Targeting APE1 endonuclease activity impairs metastasis and enhances genotoxic therapy response in pancreatic cancer
Journal of Experimental & Clinical Cancer Research · 2026
- ZNF423 depletion induces the integrated stress response and represents a potential vulnerability in NF1-associated MPNST
bioRxiv (Cold Spring Harbor Laboratory) · 2026
- Figshare×14
- Cancer Research×10
- bioRxiv (Cold Spring Harbor Laboratory)×3
- Journal of Experimental & Clinical Cancer Research×2
- British Journal of Cancer×2
- Qingbo Lu
Medicine · Indiana University
- Melissa L. Fishel
Medicine · Indiana University
- D. Wade Clapp
Medicine · Indiana University
- Steven P. Angus
Medicine · Indiana University
- Li Jiang
Medicine · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 27, 2026.
Claim or correct this profile