Publications
100
Citations
5,442
Est. group size
~3
Recurring co-author estimate
Active years
29
Publishing since 1997
This researcher studies how cystic fibrosis (a genetic disease caused by mutations in the CFTR chloride channel) affects immune cells like neutrophils and macrophages, and how bacterial infections such as Pseudomonas aeruginosa and Staphylococcus aureus interact with the immune system in this disease. Recent work also examines how CFTR-targeted drug therapies (modulators like elexacaftor/tezacaftor/ivacaftor) change immune cell function and inflammation. Prospective students would work at the intersection of immunology, microbiology, and respiratory disease research, often using lab models such as mouse models and patient-derived cells.
Publication output was steady at around 4-7 papers per year for most of the last decade, dipped sharply in 2023, then rose notably in 2024-2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Proteomics profiling of inflammatory responses to elexacaftor/tezacaftor/ivacaftor in cystic fibrosis
Frontiers in Immunology · 2025
- Flow cytometric measurement of CFTR-mediated chloride transport in human neutrophils
Journal of Leukocyte Biology · 2025
- Colistin-induced transition from non-mucoid to mucoid phenotype in resistant <i>Pseudomonas aeruginosa</i> ST3351
Virulence · 2025
- ENaC contributes to macrophage dysfunction in cystic fibrosis
American Journal of Physiology-Lung Cellular and Molecular Physiology · 2025
- CFTR mutation leads to intrinsic dysfunction in neutrophils from people with Cystic Fibrosis
bioRxiv (Cold Spring Harbor Laboratory) · 2025
- Corrigendum to Noninflammatory 97-amino acid High Mobility Group Box 1 derived polypeptide disrupts and prevents diverse biofilms. EBioMedicine 107 (2024)
EBioMedicine · 2025
- BPS2025 - Chloride intracellular channels compensate CFTR activity in macrophages
Biophysical Journal · 2025
- Author Correction: Bacterial biofilm-derived H-NS protein acts as a defense against Neutrophil Extracellular Traps (NETs)
npj Biofilms and Microbiomes · 2025
- 184 Utilizing a humanized CFTR mouse model to define neutrophilic inflammation in response to Burkholderia cenocepacia infection
Journal of Cystic Fibrosis · 2025
- High-risk Pseudomonas aeruginosa clones harboring β-lactamases: 2024 update
Heliyon · 2024
- α-Hemolysin-mediated endothelial injury contributes to the development of <i>Staphylococcus aureus</i> -induced dermonecrosis
Infection and Immunity · 2024
- ENaC contributes to macrophage dysfunction in cystic fibrosis
bioRxiv (Cold Spring Harbor Laboratory) · 2024
- High-Risk Clones Of&nbsp;<i>Pseudomonas Aeruginosa</i>: A Dangerous Combination between Virulence Factors Expression And&nbsp;Β-Lactamases Production
SSRN Electronic Journal · 2024
- Lipid a Modification in an Isolate of Pseudomonas Aeruginosa from a Child with Cystic Fibrosis Increases Host Inflammatory Immune Responses
SSRN Electronic Journal · 2024
- 34 Effect of elexacaftor/tezacaftor/ivacaftor treatment on metabolic shift of cystic fibrosis neutrophils
Journal of Cystic Fibrosis · 2024
- Journal of Cystic Fibrosis×9
- The Journal of Immunology×7
- Frontiers in Immunology×5
- Scientific Reports×3
- bioRxiv (Cold Spring Harbor Laboratory)×3
- Sankalp Malhotra
Medicine · The Ohio State University
- A. Hinton
Medicine · The Ohio State University
- A. Jay Freeman
Medicine · The Ohio State University
- Ala Shaikhkhalil
Medicine · The Ohio State University
- Alexander Weymann
Medicine · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile