Publications
30
Citations
127
Est. group size
—
Recurring co-author estimate
Active years
8
Publishing since 2019
Samuel A. Miller conducts biomedical research focused on the molecular biology of colorectal cancer, particularly how cells in the digestive system change their identity and behavior during tumor development. A central line of work examines how specific proteins (such as LSD1, CoREST2, and the signaling molecule STAT3) drive the differentiation of specialized gut cells in mucinous colorectal cancer. The work also involves computational and data-driven approaches, such as using gene-activity (transcriptomics) data to identify factors that influence disease.
Publication activity was low and intermittent through the late 2010s and early 2020s but rose sharply in 2025 and 2026, indicating a recent surge in output.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- Supplementary Table S1 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Figure S3 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Figure S1 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Figure S6 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Table S5 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Figure S2 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Table S3 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Figure S5 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Figure S4 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Table S2 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Supplementary Table S4 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2026
- Active learning framework leveraging transcriptomics identifies modulators of disease phenotypes
Science · 2025
- Supplementary Table S3 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2025
- Supplementary Figure S3 from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2025
- Data from LSD1 and CoREST2 Potentiate STAT3 Activity to Promote Enteroendocrine Cell Differentiation in Mucinous Colorectal Cancer
2025
- bioRxiv (Cold Spring Harbor Laboratory)×3
- Molecular Cancer Research×1
- Science×1
- Cancer Research×1
- Cancer Genetics×1
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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