Publications
104
Citations
4,943
Est. group size
~4
Recurring co-author estimate
Active years
42
Publishing since 1985
Ourania Andrisani studies how hepatitis B virus (HBV) interacts with liver cells to promote liver cancer (hepatocellular carcinoma), focusing on RNA-binding proteins, RNA helicases, and epigenetic regulators that control viral replication and gene silencing. Her work also examines how these molecular pathways affect drug responses, such as sensitivity to the cancer drug sorafenib. This research is aimed at understanding mechanisms of virus-driven liver cancer and identifying potential biomarkers and therapeutic targets.
Publication output has been relatively steady at a low-to-moderate level over the past decade, with a pronounced spike in 2023 followed by a decline in the most recent years.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- RNA binding protein CAPRIN1 suppresses STAT1 translation and interferon signalling to promote HBV replication
Gut · 2026
- DDX5 deficiency drives non-canonical NF-κB activation and NRF2 expression, influencing sorafenib response and hepatocellular carcinoma progression
Cell Death and Disease · 2024
- RNA Helicase DDX5 in Association With IFI16 and the Polycomb Repressive Complex 2 Silences Transcription of the Hepatitis B Virus by Interferon
Journal of Medical Virology · 2024
- RNA helicase DDX5 modulates sorafenib sensitivity in hepatocellular carcinoma via the Wnt/β-catenin–ferroptosis axis
Cell Death and Disease · 2023
- Two important players in poor-prognosis hepatocellular carcinoma: Extrachromosomal circular DNA (eccDNA) and its passenger, the oncogenic miR-17~92 locus
Hepatology · 2023
- Figure S1 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Table S2 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Table S1 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Figure S3 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Figure S2 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Supplementary Data from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Table S2 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Table S1 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Figure S1 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Figure S3 from PLK1 and HOTAIR Accelerate Proteasomal Degradation of SUZ12 and ZNF198 during Hepatitis B Virus–Induced Liver Carcinogenesis
2023
- Hepatology×4
- bioRxiv (Cold Spring Harbor Laboratory)×3
- Gut×2
- Cell Death and Disease×2
- Nature Reviews Gastroenterology & Hepatology×1
- Naimur Rahman
Medicine · Purdue University West Lafayette
- Christine Kim
Medicine · Indiana University
- Bingyu Yan
Medicine · Purdue University West Lafayette
- Balasubramanian Venkatakrishnan
Medicine · Indiana University
- Angela Patterson
Medicine · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 20, 2026.
Claim or correct this profile