Publications
138
Citations
8,367
Est. group size
—
Recurring co-author estimate
Active years
43
Publishing since 1984
Michael Rubart studies the electrical activity of the heart and the causes of abnormal heart rhythms (arrhythmias). A major focus is calmodulinopathy, a condition where mutations in the calmodulin protein disrupt calcium signaling and cause dangerous rhythm disorders, along with how the heart's nervous system and ion channels shape heartbeat regulation. The work spans genetic models in mice, drug and antisense therapies, and cell-based and light-controlled (optogenetic) approaches to studying and repairing heart tissue.
Publication activity has remained fairly steady over the last decade, averaging around three to four papers per year with a peak in 2024.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- PO-02-298 LOSS OF ADRENERGIC INHIBITION OF VOLTAGE-GATED CALCIUM CURRENTS IN CARDIAC SYMPATHETIC NEURONS OF CALMODULIN MUTANT MICE
Heart Rhythm · 2026
- Antiarrhythmic effects of mirabegron on ventricular fibrillation in Langendorff‐perfused rabbit ventricles
The Journal of Physiology · 2025
- PO-05-137 CARDIAC SYMPATHETIC HYPERACTIVITY IN A MOUSE MODEL OF A HUMAN CALMODULINOPATHY
Heart Rhythm · 2025
- Microparticle Mediated Delivery of Apelin Improves Heart Function in Post Myocardial Infarction Mice
Circulation Research · 2024
- Antisense Oligonucleotide Therapy for Calmodulinopathy
Circulation · 2024
- K <sup>+</sup> currents in ventricular cardiomyocytes of p.N98S-calmodulin mutant mice
American Journal of Physiology-Heart and Circulatory Physiology · 2024
- Insulin mitigates acute ischemia–induced atrial fibrillation and sinoatrial node dysfunction ex vivo
JCI Insight · 2024
- Abstract We099: Antisense Oligonucleotide Treatment of Calmodulinopathy
Circulation Research · 2024
- PO-02-211 THE ABUNDANCE OF MUTANT CALMODULIN PROTEIN DETERMINES THE SEVERITY OF THE ARRHYTHMIA PHENOTYPE IN A MOUSE MODEL OF A HUMAN CALMODULINOPATHY
Heart Rhythm · 2023
- Phosphodiesterase 1 inhibition underlies long QT phenotype caused by the human N98S-calmodulin mutant
Biophysical Journal · 2023
- Optogenetic Control of Engrafted Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes in Live Mice: A Proof-of-Concept Study
Cells · 2022
- Illuminating Kir channel function in Anderson–Tawil syndrome
Cardiovascular Research · 2021
- Calmodulinopathy in inherited arrhythmia syndromes
Tzu Chi Medical Journal · 2021
- Inhibition of Small-Conductance, Ca2+-Activated K+ Current by Ondansetron
Frontiers in Pharmacology · 2021
- Optogenetics: Background, Methodological Advances and Potential Applications for Cardiovascular Research and Medicine
Frontiers in Bioengineering and Biotechnology · 2020
- Heart Rhythm×7
- Circulation×4
- Biophysical Journal×4
- PMC×4
- American Journal of Physiology-Heart and Circulatory Physiology×3
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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