LabCompass

Mark H. Kaplan

Immunology and Microbiology · Indiana University

OpenAlex ORCID
Immune Cell Function and InteractionT-cell and B-cell ImmunologyAsthma and respiratory diseasesIL-33, ST2, and ILC PathwaysCytokine Signaling Pathways and Interactions

Publications

663

Citations

37,555

Est. group size

~3

Recurring co-author estimate

Active years

54

Publishing since 1973

Research summary
AI-generated

Mark H. Kaplan studies how specialized immune cells, particularly helper T cells and the signals (cytokines) they use to communicate, drive allergic and inflammatory responses in tissues such as the lung and skin. A major focus is on IL-9-producing T cells and cytokine signaling pathways (for example STAT6 and IL-4) that shape allergic airway inflammation, asthma, and anaphylaxis. His work also extends to related topics such as immune responses to SARS-CoV-2 and the immune environment of B-cell lymphomas.

Helper T cell biology and IL-9-producing T cellsCytokine signaling in allergic and airway inflammationAsthma, anaphylaxis, and lung immunityImmune responses to respiratory viral infectionTumor immune microenvironment in lymphoma

Publication activity has been steady across the decade, averaging roughly 18-28 papers per year with no clear long-term increase or decline.

Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026

Publication cadence
Publications per year over the last 10 years — averaging 18.4/year recently
2017: 28 publications172018: 28 publications182019: 35 publications35192020: 30 publications202021: 22 publications212022: 19 publications222023: 28 publications232024: 23 publications242025: 20 publications252026: 2 publications26
Recent publications
Publishes in
  • The Journal of Immunology×59
  • PMC×18
  • ImmunoHorizons×14
  • Blood×12
  • Journal of Allergy and Clinical Immunology×10

This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.

Last updated Jul 11, 2026.

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