Publications
118
Citations
6,668
Est. group size
~12
Recurring co-author estimate
Active years
22
Publishing since 2005
Kenneth L. Pitter's research focuses on pancreatic cancer, particularly how different states or subtypes of tumor cells (such as a 'basal' cell state) form, persist, and contribute to tumor growth. His work uses laboratory models of pancreatic cancer to study cell signaling pathways like WNT and Notch, which help control how cells decide their identity and behavior, and also touches on liver bile-duct cancer (cholangiocarcinoma) treatment approaches.
Publication output was relatively low and variable from 2017-2022 but spiked sharply in 2023, likely reflecting a cluster of related dataset and supplementary-file releases, with continued activity into 2024-2026.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Disruption of WNT/Notch signaling in pancreatic cancer reveals tumors depend on the intricate equilibrium of malignant cell states
Developmental Cell · 2026
- Abstract C105: Functional dissection of the basal cell state in pancreatic cancer
Cancer Research · 2024
- Multidisciplinary management in the treatment of intrahepatic cholangiocarcinoma
CA A Cancer Journal for Clinicians · 2023
- Solid tumor growth depends on an intricate equilibrium of malignant cell states
bioRxiv (Cold Spring Harbor Laboratory) · 2023
- Data from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Data from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Figure from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Figure from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Figure from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Figure from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Data from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Figure from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Figure from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Data from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- Supplementary Table from Systematic Comparison of Pancreatic Ductal Adenocarcinoma Models Identifies a Conserved Highly Plastic Basal Cell State
2023
- International Journal of Radiation Oncology*Biology*Physics×15
- Cancer Research×5
- Journal of Clinical Oncology×4
- JNCI Journal of the National Cancer Institute×2
- International Journal of Molecular Sciences×2
- Joal D. Beane
Medicine · The Ohio State University
- Trang K. Nguyen
Medicine · Indiana University
- Atilla Nakeeb
Medicine · Indiana University
- Albert Manudhane
Medicine · The Ohio State University
- A. Thyen
Medicine · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile