Publications
71
Citations
2,920
Est. group size
—
Recurring co-author estimate
Active years
23
Publishing since 2004
Kate Karelina studies how traumatic brain injury affects the brain and body over time, focusing on blood vessel and heart function, stroke risk, and metabolic changes in the brain after injury. Her work often examines how these effects differ by sex and explores mechanisms such as reduced blood flow to the brain, pericyte (blood vessel-supporting cell) dysfunction, and mitochondrial (cellular energy) stress. This research is relevant to students interested in neuroscience, cardiovascular-brain interactions, and recovery processes after brain injury or stroke.
Publication output has grown over the last decade, rising from a low of 1-3 papers per year around 2017-2021 to a higher and more consistent 4-7 papers per year from 2022-2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Traumatic brain injury impairs cardiac function independent of chronic cerebral hypoperfusion
American Journal of Physiology-Heart and Circulatory Physiology · 2026
- Abstract TP075: Circulating extracellular vesicles from the plasma of patients with silent brain infarction induce a senescence-associated secretory phenotype in human brain endothelial cells
Stroke · 2026
- Traumatic brain injury persistently increases the incidence of both ischemic and hemorrhagic strokes: Potential mechanisms
Progress in Neurobiology · 2025
- Sex‐dependent effects of a high‐fat diet‐induced obesity model on cerebrovascular health and brain metabolism
Experimental Physiology · 2025
- Aerobic training improves exercise capacity after traumatic brain injury in female, but not male, mice
Frontiers in Physiology · 2025
- Assessment of Phase-Dependent Alterations in Cortical Glycolytic and Mitochondrial Metabolism Following Ischemic Stroke
ASN NEURO · 2025
- Abstract Fri156: Cardiac Mitochondrial Dysfunction Impairs Mitochondrial Complex Activities And Induces UPR <sup>mt</sup> Stress Responses In The Cortex And Hippocampus
Circulation Research · 2025
- Cerebral hypoperfusion exacerbates vascular dysfunction after traumatic brain injury
Experimental Neurology · 2024
- Cerebral hypoperfusion exacerbates traumatic brain injury in male but not female mice
European Journal of Neuroscience · 2024
- Prospective Study of Extracellular Vesicles in Plasma as Potential Biomarkers for Silent Brain Infarcts (P10-4.013)
Neurology · 2024
- Author response for "Cerebral hypoperfusion exacerbates traumatic brain injury in male but not female mice"
2024
- Abstract Tu044: Cardiac Mitochondrial Dysfunction Induces Region-Specific Mitochondrial Stress Response In The Brain To Adapt Neuronal Changes
Circulation Research · 2024
- Pericyte dysfunction is a key mediator of the risk of cerebral ischemia
Journal of Neuroscience Research · 2023
- Exercise intensity and sex alter neurometabolic, transcriptional, and functional recovery following traumatic brain injury
Experimental Neurology · 2023
- Mild traumatic brain injury induces pericyte detachment independent of stroke vulnerability
Neuroscience Letters · 2023
- Experimental Neurology×5
- Journal of Neurotrauma×3
- European Journal of Neuroscience×2
- Brain Behavior and Immunity×2
- Neuroscience×2
- Cole Vonder Haar
Medicine · The Ohio State University
- Molly McNett
Medicine · The Ohio State University
- Matthew Pease
Medicine · Indiana University
- Elizabeth Spurlock
Medicine · The Ohio State University
- Kris M. Martens
Medicine · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile