Publications
357
Citations
15,453
Est. group size
—
Recurring co-author estimate
Active years
56
Publishing since 1971
John T. Patton conducts research in virology and infectious disease, with a focus on rotaviruses (a common cause of severe diarrhea) and how viral proteins contribute to disease and vaccine development. The publication record also includes work touching on cancer biology and immunology, such as studies of tumor cell signaling and blood cancers.
Publication activity was moderate through the late 2010s, spiked sharply in 2023, and appears lower in the most recent years, though the last few years may be incompletely indexed.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- Cluster AT Annotation Report
QUBES · 2026
- Abstract 2893: Selective inhibition of eIF4A targets tumor-associated macrophages to restore lymphoma-specific immunity
Cancer Research · 2026
- Viroporin activity is necessary for intercellular calcium signals that contribute to viral pathogenesis
Science Advances · 2025
- Stellate Ganglion Blockade for Acute Pain Management After Transoral Robotic Surgery
The Laryngoscope · 2025
- Viroporin activity from rotavirus nonstructural protein 4 induces intercellular calcium waves that contribute to pathogenesis
bioRxiv (Cold Spring Harbor Laboratory) · 2024
- Targeting the B cell receptor signaling pathway in chronic lymphocytic leukemia
Seminars in Hematology · 2024
- Rotaviruses and Rotavirus Vaccines: Special Issue Editorial
Viruses · 2024
- 49O Clinical experience of tabelecleucel in Epistein-Barr virus-positive post-transplant lymphoproliferative disease (EBV+ PTLD) involving the central nervous system
Immuno-Oncology Technology · 2023
- Supplementary Figure 3 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- Supplementary Figure 4 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- Supplementary Figure 1 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- Supplementary Figure 2 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- Supplementary Figure 4 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- Supplementary Figure 1 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- Supplementary Figure 2 from p53-Mediated Growth Suppression in Response to Nutlin-3 in Cyclin D1–Transformed Cells Occurs Independently of p21
2023
- bioRxiv (Cold Spring Harbor Laboratory)×10
- Journal of Virology×6
- Blood×4
- Journal of General Virology×3
- mBio×3
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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