LabCompass

Johannes Schlöndorff

Medicine · The Ohio State University

Established · publishing since 1993

Publications

57

Citations

4,449

Est. group size

Recurring co-author estimate

Active years

34

Publishing since 1993

Research summary
AI-generated

Johannes Schlöndorff studies the molecular causes of kidney disease, focusing on how genetic mutations affect specialized kidney filtering cells called podocytes and the proteins that support their structure (such as ACTN4, INF2, and TRPC6). This work aims to understand rare and genetic forms of kidney damage, including focal segmental glomerulosclerosis (FSGS) and disease linked to APOL1 gene variants, which are more common in people of African ancestry. The research combines cell biology, mouse models, and genetics to explain why certain gene mutations cause kidney injury.

Podocyte biology and cytoskeleton regulationGenetic causes of kidney disease (FSGS, APOL1, TRPC6, INF2)Ion channel and receptor function in the kidneyProtein trafficking and glycosylation in diseaseClinical and translational nephrology

Publication output has been relatively steady but modest over the last decade, with a peak in 2020 and an average of about 2 papers per year over the last 5 years.

Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026

Publication cadence
Publications per year over the last 10 years — averaging 2.0/year recently
2017: 1 publication172018: 1 publication182019: 4 publications192020: 8 publications8202021: 1 publication212022: 4 publications222023: 1 publication232024: 2 publications242025: 2 publications252026: 1 publication26
Recent publications
Publishes in
  • Kidney International×5
  • Journal of the American Society of Nephrology×5
  • bioRxiv (Cold Spring Harbor Laboratory)×3
  • American Journal of Transplantation×2
  • Kidney International Reports×2
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This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.

Last updated Jul 19, 2026.

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