Jenessa A. Winston
Biochemistry, Genetics and Molecular Biology · The Ohio State University
Publications
60
Citations
2,007
Est. group size
~12
Recurring co-author estimate
Active years
47
Publishing since 1980
Jenessa A. Winston studies how gut bacteria interact with bile acids (chemicals made by the liver that help digest fat) and how this relationship affects gastrointestinal disease in animals, especially infections like Clostridioides difficile. Her work spans laboratory mouse models, companion animal (dog and cat) clinical research, and studies of the gut microbiome's role in disease and recovery, including topics like fecal microbiota transplantation and diarrhea.
Publication output grew substantially from 2017 to a peak around 2022-2023, and has remained fairly steady at a high level through 2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Bile Acid Dysregulation: Pathophysiology and Therapeutic Approaches to Bile Acid Diarrhea
Veterinary Clinics of North America Small Animal Practice · 2026
- The Effects of Hygiene Standards Pre- and Post- COVID-19 Shutdown on Fever and Diarrhea Incidence in a Daycare
Ohio Journal of Public Health · 2025
- <i>Escherichia coli</i> –associated renal malakoplakia in 2 dogs
Journal of Veterinary Diagnostic Investigation · 2025
- Clinical Guidelines for Fecal Microbiota Transplantation in Companion Animals
Advances in Small Animal Care · 2024
- Collaborative Metabolism: Gut Microbes Play a Key Role in Canine and Feline Bile Acid Metabolism
Veterinary Sciences · 2024
- Secondary bile acid ursodeoxycholic acid alters weight, the gut microbiota, and the bile acid pool in conventional mice
PLoS ONE · 2021
- Ursodeoxycholic Acid (UDCA) Mitigates the Host Inflammatory Response during Clostridioides difficile Infection by Altering Gut Bile Acids
Infection and Immunity · 2020
- Clostridioides difficile carriage in animals and the associated changes in the host fecal microbiota
Anaerobe · 2020
- Cefoperazone-treated Mouse Model of Clinically-relevant <em>Clostridium difficile</em> Strain R20291
UNC Libraries · 2020
- Ursodeoxycholic acid (UDCA) mitigates the host inflammatory response during <i>Clostridioides difficile</i> infection by altering gut bile acids which attenuates NF-κB signaling via bile acid activated receptors
bioRxiv (Cold Spring Harbor Laboratory) · 2020
- Diversification of host bile acids by members of the gut microbiota
Gut Microbes · 2019
- Characterization of <i>C. difficile</i> strains isolated from companion animals and the associated changes in the host fecal microbiota
bioRxiv (Cold Spring Harbor Laboratory) · 2019
- Secondary bile acid ursodeoxycholic acid (UDCA) alters weight, the gut microbiota, and the bile acid pool in conventional mice
bioRxiv (Cold Spring Harbor Laboratory) · 2019
- Defining the Dynamics Between the Gut Microbiota, Microbial Derived Secondary Bile Acid Ursodeoxycholic Acid (UDCA), and Clostridioides difficile Pathogenesis.
NCSU Libraries Repository (North Carolina State University Libraries) · 2019
- Inhibition of spore germination, growth, and toxin activity of clinically relevant C. difficile strains by gut microbiota derived secondary bile acids
Anaerobe · 2017
- Journal of the American Veterinary Medical Association×6
- bioRxiv (Cold Spring Harbor Laboratory)×6
- American Journal of Veterinary Research×4
- Anaerobe×3
- Journal of Veterinary Diagnostic Investigation×3
- Morgan V. Evans
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Vanessa L. Hale
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Sunjun Jung
Biochemistry, Genetics and Molecular Biology · Purdue University West Lafayette
- Li Chen
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Poonam Gopika Vinayamohan
Biochemistry, Genetics and Molecular Biology · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile