Publications
201
Citations
7,240
Est. group size
~10
Recurring co-author estimate
Active years
31
Publishing since 1996
James R. Fuchs's work spans medicinal chemistry and cancer biology, including the design of chemically modified natural-product compounds (semisynthetic derivatives) and studies of cell-signaling proteins involved in cancer progression, such as CD147 and STAT3 pathways in pancreatic cancer. The bibliographic record also includes tangential contributions related to computational chemistry (retrosynthesis prediction models) and topics like HIV drug development and curcumin-based therapeutics, suggesting a broad interest in drug discovery approaches. Prospective students would likely engage with a mix of organic synthesis, molecular biology, and possibly computational methods applied to therapeutic development.
Publication output was relatively steady and modest from 2017-2022 (2-10 per year), then spiked sharply in 2023 (51 outputs, largely supplementary materials tied to a single major study) before returning to a more typical range in 2024-2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Semisynthetic derivatives of the fungal metabolite eupenifeldin via targeting the tropolone hydroxy groups
Bioorganic & Medicinal Chemistry Letters · 2024
- Supplementary Figure 2 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Tables 2 - 4 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Figure 3 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Figure 4 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Table 1 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Figure 1 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Table 5 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Figures 5 - 6 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Data from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Data from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Tables 2 - 4 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Table 1 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Figures 5 - 6 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Supplementary Figure 2 from HAb18G/CD147 Promotes pSTAT3-Mediated Pancreatic Cancer Development via CD44s
2023
- Journal of Natural Products×6
- ACS Medicinal Chemistry Letters×4
- Cancer Research×4
- mBio×3
- Journal of Biological Chemistry×3
- David W. Haas
Medicine · Indiana University
- Daniel Adu‐Ampratwum
Medicine · The Ohio State University
- Ashish Sharma
Medicine · Purdue University West Lafayette
- S. Balakrishna Pai
Medicine · Purdue University West Lafayette
- Nitin B. Charbe
Medicine · Indiana University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile