LabCompass

James A. Marrs

Biochemistry, Genetics and Molecular Biology · Indiana University

OpenAlex ORCID
Wnt/β-catenin signaling in development and cancerPrenatal Substance Exposure EffectsZebrafish Biomedical Research ApplicationsDevelopmental Biology and Gene RegulationHippo pathway signaling and YAP/TAZ

Publications

115

Citations

3,319

Est. group size

—

Recurring co-author estimate

Active years

37

Publishing since 1990

Research summary
AI-generated

James A. Marrs studies how chemicals and genetic changes affect early animal development, primarily using zebrafish (a small fish whose transparent embryos make it a popular model for studying human biology). Much of the work examines how exposures such as alcohol, opioids, pesticides, and heavy metals disrupt embryonic development and behavior, as well as how zebrafish can model human epilepsy and be used to test anti-seizure drugs. The research also explores developmental signaling pathways and cell structure.

Zebrafish models of development and diseaseDevelopmental toxicity of alcohol, drugs, and chemicalsFetal alcohol spectrum disorderEpilepsy and anti-seizure drug testingCell signaling and morphogenesis

Publication activity has been fairly steady over the past decade, averaging around three to five papers per year with some year-to-year variation.

Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026

Publication cadence
Publications per year over the last 10 years — averaging 3.2/year recently
2017: 6 publications6172018: 3 publications182019: 4 publications192020: 6 publications6202021: 4 publications212022: 5 publications222023: 1 publication232024: 5 publications242025: 3 publications252026: 2 publications26
Recent publications
Publishes in
  • Biomedicines×10
  • PMC×4
  • PLoS ONE×3
  • Neurotoxicology and Teratology×2
  • The FASEB Journal×2

This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.

Last updated Jul 11, 2026.

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