Jagannath Misra
Biochemistry, Genetics and Molecular Biology · Indiana University
Publications
53
Citations
640
Est. group size
~1
Recurring co-author estimate
Active years
49
Publishing since 1978
Jagannath Misra studies how cells respond to stress at the molecular level, focusing on pathways in the endoplasmic reticulum (the cell's protein-folding compartment) and the 'integrated stress response' that helps cells cope with adverse conditions. This work connects to diseases including prostate cancer and liver fibrosis, examining topics like how caloric restriction can improve cancer therapy and how stress-signaling proteins drive scarring in the liver.
Publication activity has been irregular but rose sharply in 2025, driven largely by a set of supplementary files from a single cancer study, indicating recent growth in output.
Generated by claude-opus-4-8 from public bibliographic data · Jul 9, 2026
Typically publishes in teams of ~16 · 8% small-team papers (≤3 authors) · across 18 venues
- The PERK–GADD45A axis is a key driver of hepatic stellate cell activation
Hepatology Communications · 2026
- Structural basis for pseudokinase-mediated regulation of GCN2 in the integrated stress response
Proceedings of the National Academy of Sciences · 2026
- FAM134B controls collagen I dynamics in hepatic stellate cell-driven fibrosis
American Journal of Physiology-Gastrointestinal and Liver Physiology · 2025
- Editorial: Nutrients, stress response, and human health
Frontiers in Nutrition · 2025
- Abstract 1412: Translational regulation of <i>MYC</i> in acute lymphoblastic leukemia
Cancer Research · 2025
- Supplementary Figure S1 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary Figure S11 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
Cancer Research · 2025
- Supplementary File 1 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary Figure S7 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary File 2 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary Figure S8 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary Figure S10 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary File 3 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Supplementary Figure S12 from Caloric Restriction Enhances the Efficacy of Antiandrogen Therapy in Prostate Cancer by Inhibiting Androgen Receptor Translation
2025
- Cancer Research×3
- Nucleic Acids Research×2
- Journal of Biological Chemistry×2
- Trends in Endocrinology and Metabolism×1
- eLife×1
- Ronald C. Wek
Biochemistry, Genetics and Molecular Biology · Indiana University
- Bei Liu
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Avery M. Runnebohm
Biochemistry, Genetics and Molecular Biology · Indiana University
- Aaron P. Kellogg
Biochemistry, Genetics and Molecular Biology · University of Michigan
- Billy Tsai
Biochemistry, Genetics and Molecular Biology · University of Michigan
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Sep 1, 2026.
Claim or correct this profile