Jack C. Yalowich
Biochemistry, Genetics and Molecular Biology · The Ohio State University
Publications
173
Citations
5,365
Est. group size
~11
Recurring co-author estimate
Active years
49
Publishing since 1978
Jack C. Yalowich's research focuses on how cancer cells respond to and resist chemotherapy drugs, particularly those that damage DNA or target enzymes like topoisomerase II. His recent work examines how natural compounds (such as curcumin) and DNA repair pathways (like the mismatch repair system) influence tumor cell sensitivity to treatment, as well as how immune cells called myeloid-derived suppressor cells affect anticancer drug activity.
Publication output has grown over the past decade, rising from a single paper in 2017 to a peak of 14 in 2023, though it appears to have leveled off in more recent years.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Supplementary Figure 2 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Data from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Data from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure 3 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure 1 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure Legends 1-2 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure 1 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure 3 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure Legends 1-2 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Supplementary Figure 2 from The Mismatch Repair System Modulates Curcumin Sensitivity through Induction of DNA Strand Breaks and Activation of G<sub>2</sub>-M Checkpoint
2023
- Abstract 1233: Myeloperoxidase (MPO) activity in myeloid-derived suppressor cells (MDSCs): effects on anticancer drug activity
Cancer Research · 2019
- Abstract 1233: Myeloperoxidase (MPO) activity in myeloid-derived suppressor cells (MDSCs): effects on anticancer drug activity
Experimental and Molecular Therapeutics · 2019
- Abstract 4792: The effects of KPT-330, a selective inhibitor of nuclear export, on nuclear topoisomerase II alpha levels and etoposide activity in human myeloid leukemia HL-60 cells
Cancer Research · 2016
- Journal of Pharmacology and Experimental Therapeutics×7
- The FASEB Journal×5
- Molecular Pharmacology×4
- ACS Medicinal Chemistry Letters×4
- Cancer Research×4
- Mark Cushman
Biochemistry, Genetics and Molecular Biology · Purdue University West Lafayette
- Chelsea A. Mann
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Yanran Lu
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Mark J. Mitton‐Fry
Biochemistry, Genetics and Molecular Biology · The Ohio State University
- Daniel E. Beck
Pharmacology, Toxicology and Pharmaceutics · Purdue University West Lafayette
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile