Haoran Huang
Neuroscience · The Ohio State University
Publications
29
Citations
417
Est. group size
—
Recurring co-author estimate
Active years
10
Publishing since 2017
This researcher studies mechanisms of nervous system injury and degeneration, including spinal cord injury, nerve fiber (axon) degeneration, neuroinflammation, and inherited neurodegenerative conditions like cerebellar ataxia. Work spans molecular pathways (such as inflammation signaling, mitochondrial stress, and calcium-related processes), cell biology, and potential therapeutic targets including natural compounds and ion channels. The publication record reflects collaborative, multi-topic contributions across neuroscience and molecular biology rather than a single narrow focus.
Publication output has grown from sporadic single papers in the late 2010s to a more consistent 4-7 papers per year since 2023, indicating an increasing and currently active publication pace.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Obacunone Promotes Functional Recovery After Spinal Cord Injury by Attenuating Neuroinflammation by Targeting the TLR4/MyD88/p38 MAPK Pathway
Drug Design Development and Therapy · 2026
- Death receptor 6 does not regulate axon degeneration and Schwann cell injury responses during Wallerian degeneration
eLife · 2026
- Death receptor 6 does not regulate axon degeneration and Schwann cell injury responses during Wallerian degeneration
eLife · 2026
- STK40 contributes to neuropathic pain by modulating macrophagic glycogen metabolism and inflammatory responses through the AKT/GSK3β/GYS1 axis
Brain Behavior and Immunity · 2026
- Bone marrow mesenchymal stem cells-derived exosomal miR-24–3p alleviates spinal cord injury by targeting MAPK9 to inhibit the JNK/c-Jun/c-Fos pathway
Archives of Biochemistry and Biophysics · 2025
- The BATF2-ATF3 axis exacerbates intervertebral disc degeneration via inducing mitochondrial dysfunction
International Immunopharmacology · 2025
- Resilience to Endoplasmic Reticulum Stress Mitigates Calcium-Dependent Membrane Hyperexcitability Underlying Late Disease Onset in SCA6
bioRxiv (Cold Spring Harbor Laboratory) · 2025
- Death receptor 6 does not regulate axon degeneration and Schwann cell injury responses during Wallerian degeneration
bioRxiv (Cold Spring Harbor Laboratory) · 2025
- Resilience to Endoplasmic Reticulum Stress Mitigates Membrane Hyperexcitability Underlying Late Disease Onset in a Murine Model of <scp>SCA6</scp>
Annals of Neurology · 2025
- Death receptor 6 does not regulate axon degeneration and Schwann cell injury responses during Wallerian degeneration
eLife · 2025
- Death receptor 6 does not regulate axon degeneration and Schwann cell injury responses during Wallerian degeneration
eLife · 2025
- Genetically encoded Nδ-vinyl histidine for the evolution of enzyme catalytic center
Nature Communications · 2024
- P2Y6R Inhibition Induces Microglial M2 Polarization by Promoting PINK1/Parkin-Dependent Mitophagy After Spinal Cord Injury
Molecular Neurobiology · 2024
- A natural agent, 5-deoxycajanin, mitigates estrogen-deficiency bone loss via modulating osteoclast-osteoblast homeostasis
International Immunopharmacology · 2024
- Targeting Ion Channels and Purkinje Neuron Intrinsic Membrane Excitability as a Therapeutic Strategy for Cerebellar Ataxia
Life · 2023
- bioRxiv (Cold Spring Harbor Laboratory)×6
- eLife×4
- Molecular Biology of the Cell×2
- Proceedings of the National Academy of Sciences×2
- Human Molecular Genetics×2
- Jing Yang
Neuroscience · Indiana University
- Quetzalli D. Angeles-López
Neuroscience · The Ohio State University
- David D. Bushart
Neuroscience · The Ohio State University
- Chad Hoyle
Neuroscience · The Ohio State University
- Deb Kegelmeyer
Neuroscience · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
Claim or correct this profile