Gonzalo Viana Di Prisco
Neuroscience · Indiana University
Publications
75
Citations
4,580
Est. group size
~5
Recurring co-author estimate
Active years
47
Publishing since 1980
Gonzalo Viana Di Prisco studies how connections between brain cells (synapses) change and adapt, a process called synaptic plasticity, and how this relates to behavior, brain injury, and addiction. His work spans molecular mechanisms of memory and drug responses (such as to cocaine and nicotine) as well as how the brain controls movement and locomotion. He also examines how factors like maternal diet and early life adversity affect brain and social development.
Publication activity has been intermittent over the past decade, with active bursts in some years (e.g., 2020 and 2022) and quieter periods, averaging about 3.6 papers per year over the last five years.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- Editorial: Methodological innovations and translational insights in Early Life Adversity studies
Frontiers in Behavioral Neuroscience · 2026
- Synaptic Plasticity in the Injured Brain Depends on the Temporal Pattern of Stimulation
Journal of Neurotrauma · 2024
- Activity of cat premotor cortex neurons during visually guided stepping
Journal of Neurophysiology · 2023
- SYNAPTIC PLASTICITY IN THE INJURED BRAIN DEPENDS ON THE TEMPORAL PATTERN OF STIMULATION
bioRxiv (Cold Spring Harbor Laboratory) · 2023
- Signals from posterior parietal area 5 to motor cortex during locomotion
Cerebral Cortex · 2022
- Microbial Reconstitution Reverses Maternal Diet-Induced Social and Synaptic Deficits in Offspring
Cell · 2016
- Translational control by eIF2α phosphorylation regulates vulnerability to the synaptic and behavioral effects of cocaine
eLife · 2016
- eIF2α-mediated translational control regulates the persistence of cocaine-induced LTP in midbrain dopamine neurons
eLife · 2016
- Translational control of nicotine-evoked synaptic potentiation in mice and neuronal responses in human smokers by eIF2α
eLife · 2016
- Author response: eIF2α-mediated translational control regulates the persistence of cocaine-induced LTP in midbrain dopamine neurons
2016
- Author response: Translational control of nicotine-evoked synaptic potentiation in mice and neuronal responses in human smokers by eIF2α
2016
- Author response: Translational control by eIF2α phosphorylation regulates vulnerability to the synaptic and behavioral effects of cocaine
2016
- eLife×4
- bioRxiv (Cold Spring Harbor Laboratory)×4
- Figshare×4
- Alzheimer s & Dementia×2
- eNeuro×2
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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