George A. Kyriazis
Neuroscience · The Ohio State University
Publications
67
Citations
1,693
Est. group size
~4
Recurring co-author estimate
Active years
53
Publishing since 1973
George A. Kyriazis studies how the body senses nutrients like sugar, focusing on sweet taste receptors (such as TAS1R2) found outside the tongue, including in skeletal muscle, and how these receptors influence metabolism, exercise response, and disease risk factors like obesity and diabetes. The work spans genetic studies in mice and humans, cellular signaling, and links between nutrient sensing and conditions such as glioblastoma and cardiac stress from diet. This research combines physiology, genetics, and metabolism to understand how nutrient-sensing receptors affect whole-body health.
Publication output has grown over the past decade, with a notable surge in 2023 followed by a return to a more moderate but steady pace in 2024-2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- The cardiac METTL3/m6A pathway regulates the systemic response to Western diet
JCI Insight · 2025
- Low TAS1R2 Sweet Taste Receptor Expression in Skeletal Muscle of Genetically Diverse BXD Mice Mirrors Transcriptomic Signatures of Loss-of-Function Mice
Nutrients · 2025
- The TAS1R2 G-protein-coupled receptor is an ambient glucose sensor in skeletal muscle that regulates NAD homeostasis and mitochondrial capacity
Nature Communications · 2024
- A partial loss-of-function variant (Ile191Val) of the TAS1R2 glucose receptor is associated with enhanced responses to exercise training in older adults with obesity: A translational study
Metabolism · 2024
- Beyond brain injury biomarkers: chemoattractants and circulating progenitor cells as biomarkers of endogenous rehabilitation effort in preterm neonates with encephalopathy
Frontiers in Pediatrics · 2023
- The TAS1R2 sweet taste receptor regulates skeletal muscle mass and fitness
Research Square · 2023
- Data from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Figure Legends 1-2 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Figure 2 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Figure 1 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Table 1 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Table 1 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Figure 1 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Figure 2 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Supplementary Figure Legends 1-2 from Receptor Channel TRPC6 Is a Key Mediator of Notch-Driven Glioblastoma Growth and Invasiveness
2023
- Molecular Metabolism×3
- American Journal of Clinical Nutrition×2
- Microbiome×1
- American Journal of Physiology-Endocrinology and Metabolism×1
- Biomedicines×1
- Robert V. Considine
Neuroscience · Indiana University
- Kimberly P. Kinzig
Neuroscience · Purdue University West Lafayette
- Joan Serrano
Nursing · The Ohio State University
- Deborah A. Swartz‐Basile
Neuroscience · Indiana University
- Eugene Choi
Neuroscience · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 19, 2026.
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