Fatema Currim
Immunology and Microbiology · Purdue University West Lafayette
Publications
23
Citations
278
Est. group size
~1
Recurring co-author estimate
Active years
6
Publishing since 2020
Fatema Currim's research examines how cells respond to stress at the molecular level, focusing on two main disease areas: Parkinson's disease and breast cancer. In Parkinson's disease work, the lab studies how nerve cells release tiny signaling packages called exosomes (carrying molecules like microRNAs) that can trigger cell death in neighboring neurons and glial cells, and how mitochondria (the cell's energy-producing structures) contribute to this damage. In breast cancer, the work explores immune signaling pathways (such as STING and NF-κB) and mitochondrial changes that influence tumor growth and metastasis.
Publication output was minimal before 2020 but has grown substantially since 2021, with a particularly active and increasing output in 2023-2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- hsa‐miR‐22‐3p‐Mediated Exosome Release From Neurons Induces Apoptosis in Recipient Glial and Neuronal Cells in Parkinson's Disease Stress Conditions
European Journal of Neuroscience · 2025
- Mitochondrial toxicants to model Parkinson’s disease
Advances in neurotoxicology · 2025
- Retraction Note: The analog of cGAMP, c-di-AMP, activates STING mediated cell death pathway in estrogen-receptor negative breast cancer cells
APOPTOSIS · 2025
- hsa-miR-22-3p mediated exosome release from neurons induces apoptosis in the recipient glial and neuronal cells in Parkinson's disease
2025
- Author response for "hsa-miR-22-3p mediated exosome release from neurons induces apoptosis in recipient glial and neuronal cells in Parkinson’s disease stress conditions"
2025
- Author response for "hsa-miR-22-3p mediated exosome release from neurons induces apoptosis in recipient glial and neuronal cells in Parkinson’s disease stress conditions"
2025
- Selective dopaminergic neurotoxicity modulated by inherent cell-type specific neurobiology
NeuroToxicology · 2024
- Enhanced translocation of <scp>TRIM32</scp> to mitochondria sensitizes dopaminergic neuronal cells to apoptosis during stress conditions in Parkinson's disease
FEBS Journal · 2024
- Mitochondrial E3 ligase TRIM71 affects mitochondrial complex assembly and sensitizes dopaminergic neuronal cells to apoptosis in Parkinson’s Disease (PD)
The International Journal of Biochemistry & Cell Biology · 2024
- TNF-α induced NF-κB mediated LYRM7 expression modulates the tumor growth and metastatic ability in breast cancer
Free Radical Biology and Medicine · 2023
- Do different exosome biogenesis pathways and selective cargo enrichment contribute to exosomal heterogeneity?
Biology of the Cell · 2023
- hsa-miR-320a mediated exosome release under PD stress conditions rescue mitochondrial ROS and cell death in the recipient neuronal and glial cells
The International Journal of Biochemistry & Cell Biology · 2023
- Author response for "Enhanced translocation of <scp>TRIM32</scp> to mitochondria sensitizes dopaminergic neuronal cells to apoptosis during stress conditions in Parkinson's disease"
2023
- DNA damage induces STING mediated IL-6-STAT3 survival pathway in triple-negative breast cancer cells and decreased survival of breast cancer patients
APOPTOSIS · 2022
- TNF-α differentially modulates subunit levels of respiratory electron transport complexes of ER/PR +ve/−ve breast cancer cells to regulate mitochondrial complex activity and tumorigenic potential
Cancer & Metabolism · 2021
- APOPTOSIS×3
- Free Radical Biology and Medicine×2
- NeuroToxicology×2
- The International Journal of Biochemistry & Cell Biology×2
- Cancer & Metabolism×1
- Adriana Forero
Immunology and Microbiology · The Ohio State University
- Adam D. Kenney
Immunology and Microbiology · The Ohio State University
- Ashley Zani
Immunology and Microbiology · The Ohio State University
- Ajit Elhance
Immunology and Microbiology · The Ohio State University
- Beiyuan Liang
Immunology and Microbiology · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 20, 2026.
Claim or correct this profile