Brittany L. Allen-Petersen
Biochemistry, Genetics and Molecular Biology · Purdue University West Lafayette
Publications
105
Citations
1,754
Est. group size
~15
Recurring co-author estimate
Active years
20
Publishing since 2007
Brittany L. Allen-Petersen studies the molecular biology of pancreatic cancer, focusing on how signaling proteins, gene mutations (such as Myc and Kras), and the tumor's surrounding support cells (fibroblasts) contribute to tumor growth, spread, and resistance to treatment. Her lab also develops laboratory models, such as mouse models and patient-derived cell cultures, and explores potential drug targets including enzyme inhibitors, to better understand and treat this disease.
Publication output has grown substantially over the last decade, rising from about 1 publication per year in 2017-2018 to over 20-29 per year in 2023 and 2025.
Generated by claude-sonnet-5 from public bibliographic data · Jul 20, 2026
- Identification, optimization, and structural elucidation of chloroacetamide scaffold as covalent inhibitors for Ubiquitin C-terminal Hydrolase L3
bioRxiv (Cold Spring Harbor Laboratory) · 2026
- The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
Cancer Research · 2025
- Protocol to purify and culture human pancreatic cancer cells from patient-derived xenografts
STAR Protocols · 2025
- Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse model
bioRxiv (Cold Spring Harbor Laboratory) · 2025
- Supplementary Dataset 9 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Dataset 14 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Dataset 10 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Dataset 7 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Figure S8 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Dataset 8 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Figure S6 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Figure S1 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Figure S5 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Dataset 6 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Supplementary Figure S7 from The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic Cancer
2025
- Cancer Research×22
- bioRxiv (Cold Spring Harbor Laboratory)×5
- Nature Communications×2
- Journal of Biological Chemistry×2
- Oncogene×2
- Erryk Katayama
Medicine · The Ohio State University
- Susan Tsai
Medicine · The Ohio State University
- Matthew S. Stratton
Medicine · The Ohio State University
- Kiet T. Nguyen
Medicine · The Ohio State University
- Abdullah Altaf
Medicine · The Ohio State University
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 20, 2026.
Claim or correct this profile