Publications
902
Citations
69,991
Est. group size
—
Recurring co-author estimate
Active years
60
Publishing since 1967
Bernardino Ghetti studies neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease, and related dementias, focusing on how proteins like tau and alpha-synuclein misfold and clump together in the brain. Much of the work identifies the detailed structures of these protein aggregates, links genetic variants to disease subtypes, and develops sensitive laboratory tests and potential treatments to detect or block these harmful protein deposits.
Publication output has been steady and high across the decade, generally 23-34 papers per year, with lower counts in 2025-2026 that likely reflect incomplete recent indexing.
Generated by claude-opus-4-8 from public bibliographic data · Jul 11, 2026
- Distinct TAF15 amyloid filament folds define multiple subtypes of FTLD-TAF15
bioRxiv (Cold Spring Harbor Laboratory) · 2026
- A same day α-synuclein RT-QuIC seed amplification assay for synucleinopathy biospecimens
npj Biosensing · 2025
- Tau filaments with the Alzheimer fold in human MAPT mutants V337M and R406W
Nature Structural & Molecular Biology · 2025
- Deciphering distinct genetic risk factors for FTLD-TDP pathological subtypes via whole-genome sequencing
Nature Communications · 2025
- Tau uptake by human neurons depends on receptor LRP1 and kinase LRRK2
The EMBO Journal · 2025
- Copy Number Variation and Haplotype Analysis of <scp>17q21.31</scp> Reveals Increased Risk Associated with Progressive Supranuclear Palsy and Gene Expression Changes in Neuronal Cells
Movement Disorders · 2025
- Celebrating neuropathology's contributions to Alzheimer's Disease Research Centers
Alzheimer s & Dementia · 2025
- Author Correction: Structures of α-synuclein filaments from multiple system atrophy
Nature · 2025
- Specific chemical determinants are central for achieving ligands for selective detection of amyloid-β deposits in Alzheimer’s disease
Australian Journal of Chemistry · 2025
- Classification of Tauopathies from Human Brain Homogenates through Salt-Modulated Tau Amplification
medRxiv · 2025
- Author Reply to Peer Reviews of Whole genome CRISPR screens identify a LRRK2-regulated pathway for extracellular tau uptake by human neurons
2025
- Seeding biosensor cell line that reproduces the Alzheimer tau fold
Journal of Biological Chemistry · 2025
- γ-Secretase modulator resistance of an aggressive Alzheimer-causing presenilin mutant can be overcome in the heterozygous patient state by a set of advanced compounds
Alzheimer s Research & Therapy · 2025
- Correction: γ‑Secretase modulator resistance of an aggressive Alzheimer‑causing presenilin mutant can be overcome in the heterozygous patient state by a set of advanced compounds
Alzheimer s Research & Therapy · 2025
- Discovery of potent inhibitors of α-synuclein aggregation using structure-based iterative learning
Nature Chemical Biology · 2024
- Alzheimer s & Dementia×36
- bioRxiv (Cold Spring Harbor Laboratory)×23
- Acta Neuropathologica×21
- Nature×13
- PMC×12
This profile was generated automatically from public scholarly data (OpenAlex). Group size and activity levels are estimates derived from co-authorship patterns.
Last updated Jul 11, 2026.
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